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Polymorphisms in the FK506 binding protein 5 gene are associated with attention deficit hyperactivity disorder and diurnal cortisol levels
Uppsala Univ, Dept Neurosci, Child & Adolescent Psychiat Unit, Uppsala, Sweden..ORCID iD: 0000-0003-1033-2618
Uppsala Univ, Dept Immunol Genet & Pathol, Genom, Uppsala, Sweden..
Uppsala Univ, Clin Res Ctr, Hosp Vastmanland, Uppsala, Sweden..ORCID iD: 0000-0002-8853-2508
Uppsala Univ, Dept Neurosci, Child & Adolescent Psychiat Unit, Uppsala, Sweden..
2015 (English)In: Acta Paediatrica, ISSN 0803-5253, E-ISSN 1651-2227, Vol. 104, no 9, p. 910-915Article in journal (Refereed) Published
Abstract [en]

AimPrevious studies have shown an association between childhood attention deficit hyperactivity disorder (ADHD) and a down-regulated hypothalamus-pituitary-adrenal axis (HPA axis) with low diurnal cortisol levels. Given the role of the FK506 binding protein 5 (FKBP5) as an important regulator of the negative feedback system of the HPA axis, we set out to investigate possible associations between single nucleotide polymorphisms (SNPs) in FKBP5 in relation to ADHD and diurnal cortisol levels. MethodsChildren with ADHD (n=81) and healthy comparisons (n=88) collected saliva four times during a regular school day for radioimmunoassay analysis of cortisol and for genotyping of five SNPs in FKBP5 (rs9296158, rs1360780, rs9470080, rs7748266 and rs9394309). ResultsWe found associations between SNP genotypes and ADHD as well as between genotypes and diurnal cortisol levels. One of these SNPs, rs9470080, was significantly associated with both ADHD and lower cortisol levels. ConclusionThis study contributes to previous findings on a down-regulated HPA axis in children with ADHD by demonstrating an association between ADHD, lower cortisol levels and SNPs of the FKBP5-gene. The relevance of these findings for the development and shaping of ADHD symptoms needs to be approached in larger samples, preferably also taking stress reactivity into consideration.

Place, publisher, year, edition, pages
WILEY , 2015. Vol. 104, no 9, p. 910-915
Keywords [en]
Attention deficit hyperactivity disorder, Cortisol, Feedback regulation, FK506 binding protein 5, Hypothalamus-pituitary-adrenal axis
National Category
Clinical Medicine
Identifiers
URN: urn:nbn:se:mdh:diva-52010DOI: 10.1111/apa.13056ISI: 000359786100021PubMedID: 26032970Scopus ID: 2-s2.0-84939271337OAI: oai:DiVA.org:mdh-52010DiVA, id: diva2:1484411
Available from: 2020-10-28 Created: 2020-10-28 Last updated: 2021-01-20Bibliographically approved

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Nilsson, Kent W.

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