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Transcription factor AP-2 beta genotype and psychosocial adversity in relation to adolescent depressive symptomatology
Uppsala Univ, Clin Res Ctr, Cent Hosp, S-72189 Vasteras, Sweden..ORCID iD: 0000-0002-8853-2508
Uppsala Univ, Clin Res Ctr, Cent Hosp, S-72189 Vasteras, Sweden.;Univ Umea Hosp, Dept Neurosurg, S-90185 Umea, Sweden..ORCID iD: 0000-0002-8258-0699
Uppsala Univ, Clin Res Ctr, Cent Hosp, S-72189 Vasteras, Sweden..
Uppsala Univ, Pharmacol Sect, Dept Neurosci, Uppsala, Sweden..
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2009 (English)In: Journal of neural transmission, ISSN 0300-9564, E-ISSN 1435-1463, Vol. 116, no 3, p. 363-370Article in journal (Refereed) Published
Abstract [en]

The aim of this study was to investigate possible interactions between the gene coding for activating protein-2 beta (AP-2 beta) and psychosocial factors to predict depressive symptoms in adolescents. Two-hundred 16- and 19-year-old adolescents from the county of Vastmanland, Sweden, were asked to complete a questionnaire, interviewed about psychosocial risk factors, and genotyped with regard to the transcription factor AP-2 beta intron 2 polymorphism. AP-2 beta genotype interacted significantly both with type of housing and parental separation to predict depressive symptoms. Individuals who were homozygous for the short AP-2 beta allele displayed higher depression scores when psychosocial adversity was taken into account. Amongst carriers of one or two copies of the long allele, there was no difference in depressive symptoms despite differences in psychosocial environments.

Place, publisher, year, edition, pages
SPRINGER WIEN , 2009. Vol. 116, no 3, p. 363-370
Keywords [en]
Depression, Adolescent, Gene, Environment, Transcription factor AP-2
National Category
Clinical Medicine
Identifiers
URN: urn:nbn:se:mdh:diva-52048DOI: 10.1007/s00702-009-0183-3ISI: 000263897900013PubMedID: 19184334Scopus ID: 2-s2.0-61849132227OAI: oai:DiVA.org:mdh-52048DiVA, id: diva2:1484363
Available from: 2020-10-28 Created: 2020-10-28 Last updated: 2021-01-20Bibliographically approved

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Nilsson, Kent W.

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